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Showing 1 to 12 of 188 entries
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Systematic Review Investigating Multi-disciplinary Team Approaches to Screening and Early Diagnosis of Dementia in Primary Care - What are the Positive and Negative Effects and Who Should Deliver It?.

Current Alzheimer research

Smith T, Cross J, Poland F, Clay F, Brookes A, Maidment I, Penhale B, Laidlaw K, Fox C.
PMID: 28891442
Curr Alzheimer Res. 2018;15(1):5-17. doi: 10.2174/1567205014666170908094931.

BACKGROUND: Primary care services frequently provide the initial contact between people with dementia and health service providers. Early diagnosis and screening programmes have been suggested as a possible strategy to improve the identification of such individuals and treatment and...

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Smith T, Cross J, Poland F, et al. Systematic Review Investigating Multi-disciplinary Team Approaches to Screening and Early Diagnosis of Dementia in Primary Care - What are the Positive and Negative Effects and Who Should Deliver It?. Curr Alzheimer Res. 2018;15(1):5-17doi: 10.2174/1567205014666170908094931.
Smith, T., Cross, J., Poland, F., Clay, F., Brookes, A., Maidment, I., Penhale, B., Laidlaw, K., & Fox, C. (2018). Systematic Review Investigating Multi-disciplinary Team Approaches to Screening and Early Diagnosis of Dementia in Primary Care - What are the Positive and Negative Effects and Who Should Deliver It?. Current Alzheimer research, 15(1), 5-17. https://doi.org/10.2174/1567205014666170908094931
Smith, Toby, et al. "Systematic Review Investigating Multi-disciplinary Team Approaches to Screening and Early Diagnosis of Dementia in Primary Care - What are the Positive and Negative Effects and Who Should Deliver It?." Current Alzheimer research vol. 15,1 (2018): 5-17. doi: https://doi.org/10.2174/1567205014666170908094931
Smith T, Cross J, Poland F, Clay F, Brookes A, Maidment I, Penhale B, Laidlaw K, Fox C. Systematic Review Investigating Multi-disciplinary Team Approaches to Screening and Early Diagnosis of Dementia in Primary Care - What are the Positive and Negative Effects and Who Should Deliver It?. Curr Alzheimer Res. 2018;15(1):5-17. doi: 10.2174/1567205014666170908094931. PMID: 28891442.
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Meet Our Associate Editor.

Current Alzheimer research

Marshall GA.
PMID: 27291395
Curr Alzheimer Res. 2016;13(8):845. doi: 10.2174/156720501308160602213059.

No abstract available.

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Marshall GA. Meet Our Associate Editor. Curr Alzheimer Res. 2016;13(8):845doi: 10.2174/156720501308160602213059.
Marshall, G. A. (2016). Meet Our Associate Editor. Current Alzheimer research, 13(8), 845. https://doi.org/10.2174/156720501308160602213059
Marshall, Gad A. "Meet Our Associate Editor." Current Alzheimer research vol. 13,8 (2016): 845. doi: https://doi.org/10.2174/156720501308160602213059
Marshall GA. Meet Our Associate Editor. Curr Alzheimer Res. 2016;13(8):845. doi: 10.2174/156720501308160602213059. PMID: 27291395.
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Recent breakthroughs in the understanding of frontotemporal lobar degeneration and its related disorders.

Current Alzheimer research

Borroni B.
PMID: 21524272
Curr Alzheimer Res. 2011 May;8(3):222-3. doi: 10.2174/156720511795563782.

No abstract available.

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Borroni B. Recent breakthroughs in the understanding of frontotemporal lobar degeneration and its related disorders. Curr Alzheimer Res. 2011;8(3):222-3doi: 10.2174/156720511795563782.
Borroni, B. (2011). Recent breakthroughs in the understanding of frontotemporal lobar degeneration and its related disorders. Current Alzheimer research, 8(3), 222-3. https://doi.org/10.2174/156720511795563782
Borroni, B. "Recent breakthroughs in the understanding of frontotemporal lobar degeneration and its related disorders." Current Alzheimer research vol. 8,3 (2011): 222-3. doi: https://doi.org/10.2174/156720511795563782
Borroni B. Recent breakthroughs in the understanding of frontotemporal lobar degeneration and its related disorders. Curr Alzheimer Res. 2011 May;8(3):222-3. doi: 10.2174/156720511795563782. PMID: 21524272.
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Regulatory chaperone complexes in neurodegenerative diseases: a perspective on therapeutic intervention.

Current Alzheimer research

Carman A, Kishinevsky S, Koren J, Luo W, Chiosis G.
PMID: 24251390
Curr Alzheimer Res. 2014 Jan;11(1):59-68. doi: 10.2174/1567205010666131119233044.

Protein folding, protein degradation, and protein stability are regulated by the molecular chaperones. Under pathogenic conditions, aberrant proteins can be dysfunctional, unregulated, or pathogenically mutated. These aberrant proteins are triaged by the chaperone network for the maintenance of cellular...

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Carman A, Kishinevsky S, Koren J, et al. Regulatory chaperone complexes in neurodegenerative diseases: a perspective on therapeutic intervention. Curr Alzheimer Res. 2014;11(1):59-68doi: 10.2174/1567205010666131119233044.
Carman, A., Kishinevsky, S., Koren, J., Luo, W., & Chiosis, G. (2014). Regulatory chaperone complexes in neurodegenerative diseases: a perspective on therapeutic intervention. Current Alzheimer research, 11(1), 59-68. https://doi.org/10.2174/1567205010666131119233044
Carman, Aaron, et al. "Regulatory chaperone complexes in neurodegenerative diseases: a perspective on therapeutic intervention." Current Alzheimer research vol. 11,1 (2014): 59-68. doi: https://doi.org/10.2174/1567205010666131119233044
Carman A, Kishinevsky S, Koren J, Luo W, Chiosis G. Regulatory chaperone complexes in neurodegenerative diseases: a perspective on therapeutic intervention. Curr Alzheimer Res. 2014 Jan;11(1):59-68. doi: 10.2174/1567205010666131119233044. PMID: 24251390.
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Natural Modulators of Amyloid-Beta Precursor Protein Processing.

Current Alzheimer research

Zhang C, Tanzi RE.
PMID: 22998566
Curr Alzheimer Res. 2012 Sep 13; Epub 2012 Sep 13.

Alzheimer�s disease (AD) is a devastating neurodegenerative disease and the primary cause of dementia, with no cure currently available. The pathogenesis of AD is believed to be primarily driven by Aβ, the principal component of senile plaques. Aβ is...

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Zhang C, Tanzi RE. Natural Modulators of Amyloid-Beta Precursor Protein Processing. Curr Alzheimer Res. 2012;
Zhang, C., & Tanzi, R. E. (2012). Natural Modulators of Amyloid-Beta Precursor Protein Processing. Current Alzheimer research, .
Zhang, C, and Tanzi, R E. "Natural Modulators of Amyloid-Beta Precursor Protein Processing." Current Alzheimer research vol. (2012).
Zhang C, Tanzi RE. Natural Modulators of Amyloid-Beta Precursor Protein Processing. Curr Alzheimer Res. 2012 Sep 13; Epub 2012 Sep 13. PMID: 22998566; PMCID: PMC3805747.
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β-Amyloid is an Immunopeptide and Alzheimer's is an Autoimmune Disease.

Current Alzheimer research

Weaver DF.
PMID: 34856900
Curr Alzheimer Res. 2021;18(11):849-857. doi: 10.2174/1567205018666211202141650.

BACKGROUND: As new biomolecular targets for Alzheimer's disease (AD) emerge, there is a tendency to regard these as mutually exclusive and in competition, culminating in declarations that since the "amyloid hypothesis is dead" it needs to be replaced by...

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Weaver DF. β-Amyloid is an Immunopeptide and Alzheimer's is an Autoimmune Disease. Curr Alzheimer Res. 2021;18(11):849-857doi: 10.2174/1567205018666211202141650.
Weaver, D. F. (2021). β-Amyloid is an Immunopeptide and Alzheimer's is an Autoimmune Disease. Current Alzheimer research, 18(11), 849-857. https://doi.org/10.2174/1567205018666211202141650
Weaver, Donald F. "β-Amyloid is an Immunopeptide and Alzheimer's is an Autoimmune Disease." Current Alzheimer research vol. 18,11 (2021): 849-857. doi: https://doi.org/10.2174/1567205018666211202141650
Weaver DF. β-Amyloid is an Immunopeptide and Alzheimer's is an Autoimmune Disease. Curr Alzheimer Res. 2021;18(11):849-857. doi: 10.2174/1567205018666211202141650. PMID: 34856900.
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Inflammation in the CNS - understanding various aspects of the pathogenesis of Alzheimer's disease.

Current Alzheimer research

Doroszkiewicz J, Mroczko P, Kulczyńska-Przybik A.
PMID: 34856902
Curr Alzheimer Res. 2021 Dec 02; doi: 10.2174/1567205018666211202143935. Epub 2021 Dec 02.

Alzheimer's disease is a progressive and deadly neurodegenerative disorder, and one of the most common causes of dementia in the world. Current, insufficiently sensitive and specific methods of early diagnosis and monitoring of this disease prompt a search for...

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Doroszkiewicz J, Mroczko P, Kulczyńska-Przybik A. Inflammation in the CNS - understanding various aspects of the pathogenesis of Alzheimer's disease. Curr Alzheimer Res. 2021;doi: 10.2174/1567205018666211202143935.
Doroszkiewicz, J., Mroczko, P., & Kulczyńska-Przybik, A. (2021). Inflammation in the CNS - understanding various aspects of the pathogenesis of Alzheimer's disease. Current Alzheimer research, . https://doi.org/10.2174/1567205018666211202143935
Doroszkiewicz, Julia, et al. "Inflammation in the CNS - understanding various aspects of the pathogenesis of Alzheimer's disease." Current Alzheimer research vol. (2021). doi: https://doi.org/10.2174/1567205018666211202143935
Doroszkiewicz J, Mroczko P, Kulczyńska-Przybik A. Inflammation in the CNS - understanding various aspects of the pathogenesis of Alzheimer's disease. Curr Alzheimer Res. 2021 Dec 02; doi: 10.2174/1567205018666211202143935. Epub 2021 Dec 02. PMID: 34856902.
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A Brief Comparative Look at Experimental Memory Editing Techniques for Cognitive Dysfunction.

Current Alzheimer research

Poon CH, Tan SZK, Sheng V, Wang S, Aquili L, Lim LW.
PMID: 34879803
Curr Alzheimer Res. 2021;18(10):841-848. doi: 10.2174/1567205018666211208142036.

Neuroscience has long sought to develop methods that can "edit" or even "erase" memories, with the aim to provide treatments for memory-related neurological and psychiatric diseases such as anxiety and addiction. Current efforts are heavily focused on modifying cognitive...

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Poon CH, Tan SZK, Sheng V, et al. A Brief Comparative Look at Experimental Memory Editing Techniques for Cognitive Dysfunction. Curr Alzheimer Res. 2021;18(10):841-848doi: 10.2174/1567205018666211208142036.
Poon, C. H., Tan, S. Z. K., Sheng, V., Wang, S., Aquili, L., & Lim, L. W. (2021). A Brief Comparative Look at Experimental Memory Editing Techniques for Cognitive Dysfunction. Current Alzheimer research, 18(10), 841-848. https://doi.org/10.2174/1567205018666211208142036
Poon, Chi H, et al. "A Brief Comparative Look at Experimental Memory Editing Techniques for Cognitive Dysfunction." Current Alzheimer research vol. 18,10 (2021): 841-848. doi: https://doi.org/10.2174/1567205018666211208142036
Poon CH, Tan SZK, Sheng V, Wang S, Aquili L, Lim LW. A Brief Comparative Look at Experimental Memory Editing Techniques for Cognitive Dysfunction. Curr Alzheimer Res. 2021;18(10):841-848. doi: 10.2174/1567205018666211208142036. PMID: 34879803.
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Extended Application of Digital Clock Drawing Test in the Evaluation of Alzheimer's Disease Based on Artificial intelligence and the Neural Basis.

Current Alzheimer research

Zheng X, Wang X, Zhang W, Li R, Liu M, Xu F, Li Y, Zheng J, Nie Z.
PMID: 34895125
Curr Alzheimer Res. 2021 Dec 10; doi: 10.2174/1567205018666211210150808. Epub 2021 Dec 10.

INTRODUCTION: This study aimed to build the supervised learning model to predict the state of cognitive impairment, Alzheimer's Disease (AD) and cognitive domains including memory, language, action, and visuospatial based on Digital Clock Drawing Test (dCDT) precisely.METHODS: 207 normal...

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Zheng X, Wang X, Zhang W, et al. Extended Application of Digital Clock Drawing Test in the Evaluation of Alzheimer's Disease Based on Artificial intelligence and the Neural Basis. Curr Alzheimer Res. 2021;doi: 10.2174/1567205018666211210150808.
Zheng, X., Wang, X., Zhang, W., Li, R., Liu, M., Xu, F., Li, Y., Zheng, J., & Nie, Z. (2021). Extended Application of Digital Clock Drawing Test in the Evaluation of Alzheimer's Disease Based on Artificial intelligence and the Neural Basis. Current Alzheimer research, . https://doi.org/10.2174/1567205018666211210150808
Zheng, Xiaoran, et al. "Extended Application of Digital Clock Drawing Test in the Evaluation of Alzheimer's Disease Based on Artificial intelligence and the Neural Basis." Current Alzheimer research vol. (2021). doi: https://doi.org/10.2174/1567205018666211210150808
Zheng X, Wang X, Zhang W, Li R, Liu M, Xu F, Li Y, Zheng J, Nie Z. Extended Application of Digital Clock Drawing Test in the Evaluation of Alzheimer's Disease Based on Artificial intelligence and the Neural Basis. Curr Alzheimer Res. 2021 Dec 10; doi: 10.2174/1567205018666211210150808. Epub 2021 Dec 10. PMID: 34895125.
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Cerebrospinal Fluid Biomarker Levels as Markers for Nursing Home Placement and Survival Time in Alzheimer's Disease.

Current Alzheimer research

Wattmo C, Blennow K, Hansson O.
PMID: 34719365
Curr Alzheimer Res. 2021;18(7):573-584. doi: 10.2174/1567205018666211022164952.

BACKGROUND: Cerebrospinal Fluid (CSF) biomarkers are associated with conversion from mild cognitive impairment to Alzheimer's Disease (AD), but their predictive value for later end-points has been less evaluated with inconsistent results.OBJECTIVE: We investigated potential relationships between CSF amyloid-βMETHODS: This...

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Wattmo C, Blennow K, Hansson O. Cerebrospinal Fluid Biomarker Levels as Markers for Nursing Home Placement and Survival Time in Alzheimer's Disease. Curr Alzheimer Res. 2021;18(7):573-584doi: 10.2174/1567205018666211022164952.
Wattmo, C., Blennow, K., & Hansson, O. (2021). Cerebrospinal Fluid Biomarker Levels as Markers for Nursing Home Placement and Survival Time in Alzheimer's Disease. Current Alzheimer research, 18(7), 573-584. https://doi.org/10.2174/1567205018666211022164952
Wattmo, Carina, et al. "Cerebrospinal Fluid Biomarker Levels as Markers for Nursing Home Placement and Survival Time in Alzheimer's Disease." Current Alzheimer research vol. 18,7 (2021): 573-584. doi: https://doi.org/10.2174/1567205018666211022164952
Wattmo C, Blennow K, Hansson O. Cerebrospinal Fluid Biomarker Levels as Markers for Nursing Home Placement and Survival Time in Alzheimer's Disease. Curr Alzheimer Res. 2021;18(7):573-584. doi: 10.2174/1567205018666211022164952. PMID: 34719365.
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Accelerated Epigenetic Aging in Peripheral Blood does not Predict Dementia Risk.

Current Alzheimer research

Fransquet PD, Lacaze P, Saffery R, Shah RC, Vryer R, Murray A, Woods RL, Ryan J.
PMID: 34429046
Curr Alzheimer Res. 2021;18(5):443-451. doi: 10.2174/1567205018666210823100721.

BACKGROUND: There is strong evidence that epigenetic age acceleration is associated with increased risk of later-life diseases and all-cause mortality. However, there is currently limited evidence that suggests accelerated epigenetic age is associated with dementia risk.OBJECTIVE: This study aims...

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Fransquet PD, Lacaze P, Saffery R, et al. Accelerated Epigenetic Aging in Peripheral Blood does not Predict Dementia Risk. Curr Alzheimer Res. 2021;18(5):443-451doi: 10.2174/1567205018666210823100721.
Fransquet, P. D., Lacaze, P., Saffery, R., Shah, R. C., Vryer, R., Murray, A., Woods, R. L., & Ryan, J. (2021). Accelerated Epigenetic Aging in Peripheral Blood does not Predict Dementia Risk. Current Alzheimer research, 18(5), 443-451. https://doi.org/10.2174/1567205018666210823100721
Fransquet, P D, et al. "Accelerated Epigenetic Aging in Peripheral Blood does not Predict Dementia Risk." Current Alzheimer research vol. 18,5 (2021): 443-451. doi: https://doi.org/10.2174/1567205018666210823100721
Fransquet PD, Lacaze P, Saffery R, Shah RC, Vryer R, Murray A, Woods RL, Ryan J. Accelerated Epigenetic Aging in Peripheral Blood does not Predict Dementia Risk. Curr Alzheimer Res. 2021;18(5):443-451. doi: 10.2174/1567205018666210823100721. PMID: 34429046.
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Neutrophil Granule Proteins Inhibit Amyloid Beta Aggregation and Neurotoxicity.

Current Alzheimer research

Kasus-Jacobi A, Washburn JL, Land CA, Pereira HA.
PMID: 34429047
Curr Alzheimer Res. 2021;18(5):414-427. doi: 10.2174/1567205018666210823095044.

BACKGROUND: A role for neutrophils in the pathogenesis of Alzheimer's disease (AD) is emerging. We previously showed that the neutrophil granule proteins cationic antimicrobial protein of 37 kDa (CAP37), cathepsin G (CG), and neutrophil elastase (NE) directly bind the...

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Kasus-Jacobi A, Washburn JL, Land CA, et al. Neutrophil Granule Proteins Inhibit Amyloid Beta Aggregation and Neurotoxicity. Curr Alzheimer Res. 2021;18(5):414-427doi: 10.2174/1567205018666210823095044.
Kasus-Jacobi, A., Washburn, J. L., Land, C. A., & Pereira, H. A. (2021). Neutrophil Granule Proteins Inhibit Amyloid Beta Aggregation and Neurotoxicity. Current Alzheimer research, 18(5), 414-427. https://doi.org/10.2174/1567205018666210823095044
Kasus-Jacobi, Anne, et al. "Neutrophil Granule Proteins Inhibit Amyloid Beta Aggregation and Neurotoxicity." Current Alzheimer research vol. 18,5 (2021): 414-427. doi: https://doi.org/10.2174/1567205018666210823095044
Kasus-Jacobi A, Washburn JL, Land CA, Pereira HA. Neutrophil Granule Proteins Inhibit Amyloid Beta Aggregation and Neurotoxicity. Curr Alzheimer Res. 2021;18(5):414-427. doi: 10.2174/1567205018666210823095044. PMID: 34429047.
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Showing 1 to 12 of 188 entries